Can A Keto Diet Cause High Cholesterol – According to the World Health Organization, cardiovascular disease (also known as heart disease) is the leading cause of death. The number one life in the world.
On the surface, this seems like a scary sign for humans, but there is sweetness in this bitter news when we know that heart disease is preventable and, in some cases, reversible.
Can A Keto Diet Cause High Cholesterol
Heart disease is a group of disorders of the heart and blood vessels. In general, they fall into these categories:
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The most common, preventable, and reversible type of heart disease is coronary heart disease or coronary artery disease. This occurs when the arteries that supply blood to the heart muscle harden and narrow as plaque accumulates on their inner walls. The technical term for this is atherosclerosis.
As atherosclerosis worsens, less blood is allowed to flow through the arteries. Therefore, the heart muscle cannot get the blood or oxygen it needs. This can lead to chest pain (angina) or a heart attack.
Most heart attacks occur when a blood clot suddenly cuts off the heart’s blood supply, causing permanent heart damage. These stones usually form at the site of a ruptured arterial plaque.
Over time, atherosclerosis can also weaken the heart muscle and cause heart failure and changes in the normal heart rhythm.
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Simply put, coronary artery disease is a mild process that reduces the function of the cardiovascular system and sets the stage for painful events, such as heart attacks and strokes. Once we create such conditions, however, it may seem like there isn’t much you can do to prevent or reverse them.
In fact, the opposite is true. Diet and lifestyle play an important role in preventing further plaque formation, with the potential to help us reduce atherosclerosis as well.
Although there are many genetic factors that determine the risk of heart disease, it is also preventable and reversible. This is because the most common cause of heart disease – atherosclerosis – is caused by modifiable factors (i.e., elements that are under our control).
However, don’t let the fact that atherosclerosis is the buildup of fatty plaque in your arteries fool you. This does not mean that fat clogs your arteries. In fact, fat is not the problem.
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Interestingly enough, atherosclerosis is not the problem – it is the immediate solution to the problem caused by oxidized LDL cholesterol.
LDL (low-density lipoprotein) is commonly called “bad cholesterol”, but it is actually important for our survival.
You can think of LDL as a superhero with a very short temper. LDL carries essential nutrients, such as cholesterol and vitamins, to our cells. Unfortunately, if LDL interacts with free radicals and volatile molecules in the blood, it will begin to oxidize.
Now the LDL oxidizes into a mess that causes damage to the endothelial cells (the cells that line the inside of our blood vessels). Endothelial damage activates macrophages (cells of the immune system) to neutralize oxidized LDL and prevent further damage.
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The macrophage persuades the oxidized LDL into its superhero suit and becomes the non-toxic fatty plaque that resides between the walls of blood vessels.
Our body creates this plaque to keep oxidized LDL from damaging more cells, which is why atherosclerosis is part of the (short-term) solution. The direct payoff is that oxidized LDL cannot harm healthy cells.
In the long run, however, this short-sighted solution turns out to be bad, increasing the risk of heart disease, heart attack, and stroke if we don’t make the right changes to our diet and lifestyle.
The involvement of LDL in the process of plaque formation is the main reason why it becomes a monster particle. However, so many factors play a role in increasing the risk of heart disease that just reducing the amount of LDL is not enough. This is evidenced by the fact that the disease often develops in the presence of low LDL levels as well.
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Focusing only on LDL to assess heart disease risk is like blaming the water when your drain is clogged. You can keep the sink from backing up by reducing the flow of water through the drain, but doing so does not address other problems that cause “clogging.”
To better assess the risk of cardiovascular disease, we must note these important risk factors that contribute to atherosclerosis:
Of these eleven risk factors, studies indicate that we have the most control that has the greatest impact on heart disease. One observational study of 52 countries, for example, found eight modifiable risk factors – such as abnormal blood lipids, smoking, high blood pressure, diabetes, obesity, psychological factors, consumption of fruits, vegetables, and alcohol, and regular physical activity. – covers the majority of the risk of heart attack worldwide in both sexes and all ages The living continent.
This is a remarkable finding because it shows that one of the most life-threatening complications of atherosclerosis, heart attack, is associated with globally modifiable risk factors.
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In addition, six of these eight risk factors identified in the study – i.e. Abnormal blood lipids, high blood pressure, type 2 diabetes, obesity, certain psychological factors, and consumption of fruits, vegetables, and alcohol – all can be addressed by following. The right food. The keto diet, for example, has been found to improve all six. Below, you can refer to the image for the summary version:
Because it focuses on fat consumption, the keto diet is relevant to many doctors, nutritionists, and health advocates. However, the truth about what happens when we eat more fat and cut carbs is more complicated.
Simply put, several studies indicate that the keto diet can help most people reduce their risk of heart disease while a small population may significantly increase their risk by following a standard keto diet.
To help us decipher between the two and develop a deeper understanding of what keto can do for heart disease, let’s take a look at how keto affects six risk factors that can be addressed with diet.
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The most widely studied lipids are low-density lipoprotein cholesterol (LDL), high-density lipoprotein cholesterol (HDL), and triglycerides. There are other lipid particles that can give us a more complete picture, but information about them is too scarce at this point.
Out of all the risk factors for heart disease, the effect that keto has on cholesterol levels attracts the most controversy.
Many doctors, nutritionists, and food researchers will immediately reject a diet that supports high fat consumption. For them, the possibility of damage is obvious.
More fat means more saturated fat, which typically increases LDL cholesterol levels and the risk of heart disease. Since the keto diet is one of the most high-fat diets, the link between keto and heart disease is one of the most publicized risks of the diet.
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On the other hand, low-carb doctors, dietitians, and researchers argue that keto-induced increases in cholesterol reflect an increase in cholesterol levels. This is based on an abundance of low carb and keto diet research that shows:
All in all, this is strong evidence that the carbohydrate restriction emphasized in the ketogenic diet can be a heart-healthy choice.
However, this does not mean that the “keto is bad for the heart” argument is completely wrong. A small population may be worse off, from a heart health standpoint, when they eat a high-fat, low-carbohydrate diet.
In particular, a high-fat, low-carbohydrate diet may not be a healthy choice for people with one or more of the following conditions:
Keto Diet And Cholesterol
The easiest, most accessible way to figure out that the keto diet might not be right for you is if your total cholesterol to HDL ratio is higher than 4 and/or your LDL-P (not LDL-C) is still high or rising. After starting the keto diet. If this is the case for you, it may indicate that your specific conditions require you to adapt to your keto diet (eg, follow the keto Mediterranean diet as we will learn later) or follow a different diet altogether.
For a closer look at the keto diet and cholesterol research and what this means for you, check out our keto and cholesterol article.
In contrast to cholesterol, there is very little controversy about how diet affects triglyceride levels. In fact, a meta-analysis showed a strong relationship between carb consumption and triglycerides in the blood: for every 5% decrease in total fat, triglyceride levels are expected to increase by 6%.
In addition, researchers also discovered that every 1% isoenergetic (the same amount of calories) replacement of carbohydrates with each type of fatty acid (saturated, monounsaturated, and polyunsaturated), there is a predictable decrease in triglyceride levels. The most important reduction is found in polyunsaturated fat (which is an important part of the Mediterranean diet).
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It also doesn’t matter if the carbohydrates come from table sugar or whole foods. Substituting any carbohydrate with the same amount of calories from fat lowers triglyceride levels. In addition, these results may be better for people with type 2 diabetes – a condition that can be improved by reducing carbohydrate consumption.
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