Weight Loss Clinic Near Me Phentermine – Check out all the locations below, see clinic hours and schedule an initial weight loss consultation at a time that works for you.
Select the location closest to you on the map above to view clinic hours and schedule a Medical Weight Loss Clinic exam at a time that is convenient for you.
Weight Loss Clinic Near Me Phentermine
Ann Arbor • Brighton • Canton • Chesterfield Township • Clinton Township • Dearborn / Dearborn Heights • Farmington Hills • Flint • Grand Rapids • Grandville • Grosse Pointe Woods • Jackson • Kalamazoo • Lansing • Livonia • Novi • Okemos • Rochester Hills • Royal Oak • Saginaw • Shelby City • Southfield • Southgate • Troy • Warren • White Lake
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When you enroll in the Medical Weight Loss Clinic program, the length of your program will be determined by you and the staff based on the weight you want to lose and how quickly you want to lose it. Weight loss will be determined by many factors, including, but not limited to: your medical conditions, the plan you choose, adherence to the meal plan and use of nutritional supplements. Details of your goals and program duration will be provided in writing at the time of registration. As your program progresses, your weight loss rate may change and you will have the opportunity to discuss this at each daily visit with your weight loss consultant. body, where changes to your diet plan and / or program can be made. Phentermine is an oral sympathomimetic. amine used as a short-term (eg, 8-12 weeks) adjunct in the treatment of obesity. The pharmacologic effects of phentermine are similar to amphetamines. Phentermine resin complex was approved by the FDA in 1959, but is no longer sold in the US. Phentermine hydrochloride was approved by the FDA in 1973. In the mid-90s, there was renewed interest in phentermine along with another anorectic, fenfluramine, for the treatment of obesity and substance abuse, although however, little scientific data supports this approach. On July 8, 1997, the FDA issued a ‘Dear Health Care Professional’ letter warning physicians about the development of valvular heart disease and pulmonary hypertension in women receiving a combination of fenfluramine and phentermine; fenfluramine was later withdrawn from the US market in the fall of 1997. The use of phentermine and other anorectic agents for obesity has not been evaluated and is not recommended. In May 2011, the FDA approved phentermine hydrochloride oral tablet (Suprenza) for the treatment of morbid obesity.
Little information is available in the reference texts about the mechanism of action of this drug. Phentermine is an analog of methamphetamine. Similar to amphetamines, phentermine increases the release of norepinephrine and dopamine from nerve centers and prevents their reuptake. Therefore, phentermine is classified as an indirect sympathomimetic.2 Other effects include a weak ability to raise serotonin levels in a dose-dependent manner, although the serotonin effect appears to be stronger than that of methamphetamine itself. pressure. Appetite suppression is believed to occur through direct stimulation of the satiety center in the hypothalamic and limbic regions.
Tolerance to the anorexiant effects of phentermine usually develops within a few weeks of starting treatment. The mechanism of tolerance appears to be pharmacodynamic in nature; higher doses of phentermine are needed to produce the same response. When tolerance develops to the effects of the anorexiant, it is generally recommended that phentermine be discontinued rather than the dose increased.
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Phentermine is administered orally. The rate and magnitude of exposure to phentermine under fasting conditions are similar regardless of the oral route of administration.1
Limited data are available on the pharmacokinetics of phentermine. Phentermine is mainly excreted by the kidneys. The half-life is 19-24 hours and is influenced by the pH of the urine. Because the pKa of phentermine is 9.84, the half-life is reduced to about 7-8 hours under acidic conditions of the urine.
Oral Route: Following oral administration, the major absorption of phentermine occurs from the small intestine. The duration of action after taking 8 mg capsules or tablets can be 4 hours and 12-14 hours after administration of 30 mg capsules or 37.5 mg tablets.
Phentermine Oral disintegrating tablet (ODT) reaches peak concentrations (Cmax) 3-4.4 hours after administration. Water intake prior to ODT ingestion did not affect AUC. Despite the decrease in Cmax (approximately 5%) and AUC (approximately 12%) when phentermine ODT was administered after a high-fat/high-fat breakfast, phentermine ODT can be administered with or without food. Cmax and AUC decreased by approximately 7% and 8%, respectively, when ODT was swallowed without first diluting.1
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Renal Impairment: Use with caution in patients with renal impairment. Cumulative urinary excretion of phentermine under uncontrolled urinary pH conditions is 62-85%, and increased exposure can be expected in patients with renal impairment.1
According to the manufacturers of phentermine capsules and tablets, its products are contraindicated in patients with heart disease, advanced arteriosclerosis, moderate to severe depression, nervous states or glaucoma. disease including coronary artery disease, stroke, heart attack, heart failure, and uncontrolled hypertension.5 Valvular heart disease has been reported in women receiving a combination of fenfluramine and phentermine; The safety and efficacy of combined treatment with phentermine and any drug products for weight loss, including selective serotonin reuptake inhibitors (eg, fluoxetine, sertraline, fluvoxamine, paroxetine), is not not yet established. Therefore, the combined administration of these drug products for weight loss is not recommended. Additionally, primary pulmonary hypertension (PPH) has been reported to occur in patients receiving a combination of phentermine and fenfluramine or dexfenfluramine. The possibility of an association between the use of phentermine alone and PPH or valvular heart disease cannot be excluded. The first symptom of PPH is usually dyspnea. Other early symptoms include: angina pectoris, syncope, or lower back edema. Patients should be advised to immediately report any deterioration in exercise tolerance. Treatment should be discontinued in patients who develop new, unexplained symptoms of dyspnea, angina pectoris, syncope, or low-grade edema.
Because phentermine is a sympathomimetic agent, it is contraindicated in patients with hyperthyroidism. It should also be used with caution in patients with thyroid disease.
Phentermine is contraindicated for use during or within 14 days after using MAOI therapy or other drugs with MAO-inhibiting activity. Monoamine oxidase inhibitors (MAOIs), or drugs with MAO-inhibiting activity such as furazolidone or procarbazine, may prolong and potentiate the cardiac stimulation and vasopressor effects of phentermine.4
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Phentermine is contraindicated in patients with delirious conditions, exacerbating these effects or causing an adverse drug reaction.4 Symptoms of chronic alcoholism include insomnia, irritability, personality changes, and psychological symptoms of may not be distinguished from other mental illnesses, such as schizophrenia. Phentermine can worsen certain mental conditions, such as in patients who exhibit severe or disturbed behavior, including psychosis, mania, or severe anxiety.
The use of phentermine can cause dizziness, mask the symptoms of fatigue or the need to rest, or impair the patient’s ability to participate in activities that require mental alertness. Advise patients to be careful when driving or using machines, or doing other activities that require mental alertness until they understand how the treatment will affect their mental and/or motor function. In general, drinking ethanol can worsen these effects or cause an adverse drug reaction.4 Advise patients to avoid alcohol while taking phentermine.
Use phentermine with caution in patients with diabetes. Insulin or other antidiabetic medication requirements may be altered in these patients when using phentermine during weight loss and as a result of altered dietary patterns. Patients should monitor their blood sugar regularly and follow their healthcare provider’s recommendations.5
Appetite suppression therapy is not recommended for use in patients with a history of anorexia nervosa or other eating disorders. The use of phentermine is contraindicated in patients with a known history of drug or drug abuse. Phentermine is chemically and pharmacologically related to the widely abused amphetamines. The possibility of phentermine abuse should be kept in mind when considering the desire to include the drug as part of a weight loss program. A reasonable minimum amount should be prescribed or given at one time to reduce the potential for drug abuse or diversion.5
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Phentermine products are now classified as FDA pregnancy risk X, like many anorexiants used for weight loss, and are contraindicated during pregnancy.56 The safe use of phentermine during pregnancy when not yet established; There is no known indication for the use of phentermine during pregnancy. Phentermine should not be taken by pregnant women or women who may become pregnant unless, in the doctor’s opinion, the potential benefits outweigh the potential risks.6
Sudden discontinuation of phentermine after a long high dose may result in severe depression or extreme fatigue; Changes in sleep EEG have also been observed. It is recommended to stop treatment gradually. If immediate discontinuation is medically necessary, careful monitoring and symptom control is necessary. 4
Phentermine is contraindicated during breastfeeding.5 It is not known whether phentermine and its metabolites are excreted in breast milk;
